Kari byon naked-Saints and Sages of Kashmir

Lal Ded was born in A. She is believed to be a Parmahamsa by her devotees. Studying Her works one is convinced that She is indeed a Parmahamsa. Lal Ded Rupa Bhawani regarded her as a supreme guru: Lal man Lal paramagwaram Parmanand : Unique in her yoga of dwadashanta mandala, Realizing anahata, nada-bindu and Om, Lalleshwari attained to the Supreme Ananda. The books that will be used are: 1.

Kari byon naked

Kari byon naked

VEGF -A expression decreased shortly after the dox treatment only in some tissues. However, there were increased levels of serum VEGF in the LN group, especially in patients with active nephritis as compared to quiescent nephritis group. However the intensity increased with Karl clinical size of cancer and from palpable lymph node to a tender and Kari byon naked lymph node. However, very few studies have examined whether gender differences exist in BMSC function. My fame as Lalla naker afar. The Nameless Unknown, it seems to me, Is the source of the something that we see. Who dozes off?

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The inadequate transport of drugs into the tumor tissue caused by its abnormal vasculature is a major obstacle to the treatment of cancer.

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Lal Ded was born in A. She is believed to be a Parmahamsa by her devotees. Studying Her works one is convinced that She is indeed a Parmahamsa. Lal Ded Rupa Bhawani regarded her as a supreme guru: Lal man Lal paramagwaram Parmanand : Unique in her yoga of dwadashanta mandala, Realizing anahata, nada-bindu and Om, Lalleshwari attained to the Supreme Ananda.

The books that will be used are: 1. He has the same number of vakhs in the same order as JK. Also, his vakhs are in Devnagri script which is then transliterated in English by us. In few cases the translation is from other authors, in which case it will be pointed out. Lionel D. Barnett Litt. XVII, London Wanchoo and English by Sarwanand Chaaragi, Lal Ded was far above the realm of being a Realized Soul.

This is why a title of a Parmahamsa is just a word or a phrase being used to understand where She is coming from. For the pragmatic thinkers among us please consider this question for it will tells you about Lal Ded. It is a fact that Lal Ded did not say these Vakhs for the sake of preaching, or taking the position of a teacher or one sermonizing us. She would often speak to Herself and teach Herself as will be clear from the Vakhs. Applying the usual Vedantist reasoning Who is the Speaker of the Vakh and to whom?

In many ways She answers this question Herself in Her last Vakh. Also, this tells us that people of her stature are born with Self awareness only a little rubbing is needed for them to manifest their Divinity.

I believe that They are Put in such a position for the benefit of humanity at large. Since I do not have as yet these references please try to cross check the Kashmiri with the translation and transliteration. Note the sound A is equal to the stressed sound on a is equal to aa, eg. Courtesy : Bhaskar Razdan 1.

With a rope of loose-spun thread am I towing my boat upon the sea. Would that God heard my prayer and brought me safe across!

Like water in cups of unbaked clay I run to waste. Would God I were to reach my home! Note: She compares Herself with unbaked clay which slowly wastes away all that one has earned, easily mixing with material nature rather than being above it or in control of it.

Thus, She pleads with God with pique in heart to take Her across the sea to Her real home. Though you cling to them with the anchor of steel, Not even the shadow of the things you love Will go with you when you are dead. Why then have you forgot your own true Self?

Many of the Buddhist symbols and ideas will show up in these verses, 'Vakhs'. The idea of Buddhist clinging appears here. Recall that Kashmir was one of the major centers from where Buddhism spread through out Asia. Reminds me of a story I read on the net some time ago.

A Zen master is asked what is the secret of his success. After much persistence by the student, one day the master asks him to get a thick long steel chain and he demonstrates to his student by trying to chain himself to a huge pillar. The student does not understand, the master explains; stop clinging to the world like this demonstration. Note that the true Self is the Reality only one has forgotten It. By not clinging to the unreal or that which is not permanent does the true Self dawn upon us.

Pray, Sir, how can you bring yourself to dance? See, the riches you are amassing here, nothing of them will go with you. Pray, Sir, how can you relish your food and drink? Parmahamsa RamaKrishna says that God which has cast a net in the ocean of the world is waiting to draw the net anytime, this is how He Plays this Game of Maya. He says some fishes by nature are so clever that they are never caught in this net. They are the nitya siddhas. Naradh is an example of this. The class which Lal Ded is referring here is the worldly class who hide deeper into worldliness, i.

To wake these people up Lal Ded has really addressed this Vakh 3 to them. It is not just enough to understand or be aware of the problem we humans face but also to feel this reality so much that it is difficult to swallow any food or drink.

This kind of renunciation can be sensed in the next few versus as well. And so I find me still on the embarkment, not having gone even half the way, And the day is done, the light has failed. I search my pockets but not a cowrie find: What shall I pay for the ferry fee? Just as in any field of study without putting effort one cannot expect to get results. Vivekananda says it took Him a life time of practice to gain the little he learnt.

The problem is to separate the real from the unreal, who we truly are from what we are not, keeping the inner peace and equanimity all the time. The consciousness has as if got mixed with what one is not. Parmahamsa RamaKrishna Says keep churning the buttermilk until butter is formed and it floats but does not get mixed with water.

After reaching this state does one see that what we are not is also a part of that Self. This can be done either through bhakti, jnana, raja, or karma yoga. Had all together pulled on the rope, Why should the Eleventh have lost the cow? Why should the soul have gone astray? Lal Ded is showing us how to control the mind. If one feels for the Lord by all the senses five of them and by their five activities total 10 as well towards the Lord, automatically giving the Lord control over the 11th the mind.

This should remind you of Arjun and Lord Krishna in the Chariot with five pair of horses. I feel, the same is achieved by chanting where one does not distinguish between the Lord and His Name and also one ignores or weeds out all other unnecessary thoughts Teachings of Lord Chaitanya Maha Prabhu B. Whence we come, thither we go, For ever in the round of birth and death, From nothingness to nothingness.

But sure, a mystery here abides, A Something is there for us to know. It cannot all be meaningless. Nothing endures. The nothingness in verse 7.

Whence I have come and by which way, I do not know. Wither I shall go and by which way, I do not know. Were I to know the end of it all And gain the knowledge of the truth, it would be well, for otherwise Life here is but an empty breath. I have seen a learned man die of hunger, A sere leaf drop in winter wind; I have seen an utter fool beat his cook who could not make a toothsome dish. Since then I, Lalla, anxiously await The day when the lure of the world will fall away.

Now I saw a bush in bloom; Now neither rose nor thorn was seen. Now I saw the Pandava Mother, Now she was but a potters' aunt Kawa zana kAsiy maranann shaenkh A royal fly-whisk, sunshade, chariot and throne, Merry revels, pleasures of the theater, a bed of cotton down- Which of these, you think, will go with you when you are dead?

How then can you dispel the fear of death? Why have you sunk deep in the sea of the illusory pleasures of the world? Why have you pulled down the high-banked road which could have led you safe across? The dense darkness of tamas surrounds you now, and, at the appointed time, Yama's apparitors prepare to drag your body bleeding to death.

Who can dispel your fear of death? Why do you dote upon someone, my Soul, who is not your true love? Why have you taken the false for the true? Why can't you understand, why can't you know? It is ignorance that binds you to the false, To the ever-recurring wheel of birth and death, this coming and going. You cannot tow your boat with it. What God has written "in karma's line" Cannot be altered or reversed.

God decrees reward or punishment not arbitrarily as one's 'fate', but according to ones karma. Ceremonial rites, pujas and yajnas and the like are 'rope of sand' and will not avail to change what God has decreed; for man must take the consequence of karma. However, if one is repentant of ones mistakes and does not repeat them then the prayers japa have the power of reducing the effect of karma to a large extent.

The role of an umbrella prayer in the scorching sun is an example and karma being reduced to a pin prick instead of breaking a leg is another. A Vedantin like Swami Vivekananda would say 'you are It' nothing can touch you so be brave and face your karma by watching the external as one watches a movie.

You had but tanned a carcass hide, shaped and stretched it taut on pegs, Your only care your own body which you pegged to the bonds of desire. But counsel to a fool is labour lost, Like a ball thrown at a big-sized pillar, rebounding but not hitting the mark; Or fruitless as feeding a tawny bullock on sweet molasses, And expect a yield of milk from him.

A common belief that a child resolves thus in his mother's womb.

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The inadequate transport of drugs into the tumor tissue caused by its abnormal vasculature is a major obstacle to the treatment of cancer. Anti-vascular endothelial growth factor anti- VEGF drugs can cause phenotypic alteration and maturation of the tumor's vasculature.

However, whether this consistently improves delivery and subsequent response to therapy is still controversial.

Clinical results indicate that not all patients benefit from antiangiogenic treatment, necessitating the development of criteria to predict the effect of these agents in individual tumors.

We demonstrate that, in anti- VEGF -refractory murine tumors, vascular changes after VEGF ablation result in reduced delivery leading to therapeutic failure. In these tumors, the impaired response after anti- VEGF treatment is directly linked to strong deposition of fibrillar extracellular matrix ECM components and high expression of lysyl oxidases.

The resulting condensed, highly crosslinked ECM impeded drug permeation, protecting tumor cells from exposure to small-molecule drugs.

The reduced vascular density after anti- VEGF treatment further decreased delivery in these tumors, an effect not compensated by the improved vessel quality. Pharmacological inhibition of lysyl oxidases improved drug delivery in various tumor models and reversed the negative effect of VEGF ablation on drug delivery and therapeutic response in anti- VEGF -resistant tumors.

In conclusion, the vascular changes after anti- VEGF therapy can have a context-dependent negative impact on overall therapeutic efficacy. Our results reveal the prospect to revert a possible negative effect and to potentiate responsiveness to antiangiogenic therapy by concomitantly targeting ECM-modifying enzymes.

PubMed Central. Anderson, Sean M. Luisa; Segura, Tatiana. VEGF has been incorporated into biomaterials through encapsulation, electrostatic sequestration, and covalent attachment, but the effect of these immobilization strategies on VEGF signaling has not been thoroughly investigated. Further, although growth factor internalization along with the receptor generally occurs in a physiological setting, whether this internalization is needed for receptor phosphorylation is not entirely clear.

Here we show that VEGF covalently bound through a modified heparin molecule elicits an extended response of pVEGFR-2 in human umbilical vein endothelial cells HUVECs and that the covalent linkage reduces internalization of the growth factor during receptor endocytosis. Here, mathematical models support the biological conclusions, further suggesting that VEGF internalization is significantly reduced when covalently bound, and indicating that VEGF is available for repeated phosphorylation events.

Ablation of cholesterol biosynthesis in neural stem cells increases their VEGF expression and angiogenesis but causes neuron apoptosis. Although sufficient cholesterol supply is known to be crucial for neurons in the developing mammalian brain, the cholesterol requirement of neural stem and progenitor cells in the embryonic central nervous system has not been addressed. Here we have conditionally ablated the activity of squalene synthase SQS , a key enzyme for endogenous cholesterol production, in the neural stem and progenitor cells of the ventricular zone VZ of the embryonic mouse brain.

Mutant embryos exhibited a reduced brain size due to the atrophy of the neuronal layers, and died at birth. Analyses of the E Interestingly, the neural stem and progenitor cells of the VZ, the primary target of SQS inactivation, did not undergo significant apoptosis.

Instead, vascular endothelial growth factor VEGF expression in these cells was strongly upregulated via a hypoxia-inducible factorindependent pathway, and angiogenesis in the VZ was increased. Consistent with an increased supply of lipoproteins to these cells, the level of lipid droplets containing triacylglycerides with unsaturated fatty acyl chains was found to be elevated.

Our study establishes a direct link between intracellular cholesterol levels, VEGF expression, and angiogenesis. Moreover, our data reveal a hitherto unknown compensatory process by which the neural stem and progenitor cells of the developing mammalian brain evade the detrimental consequences of impaired endogenous cholesterol biosynthesis. Instead, vascular endothelial growth factor VEGF expression in these cells was strongly upregulated via a hypoxia-inducible factor-1—independent pathway, and angiogenesis in the VZ was increased.

Crisostomo, Paul R. Concomitant pro- and anti-inflammatory properties of bone marrow stem cells BMSC may be an important aspect of their ability to heal injured tissue.

However, very few studies have examined whether gender differences exist in BMSC function. Poly lactic-co-glycolide polymer constructs cross- linked with human BMP-6 and VEGF protein significantly enhance rat mandible defect repair. We have previously shown that the combined delivery of mesenchymal stem cells MSCs , vascular endothelial growth factor VEGF and bone morphogenetic protein 6 BMP-6 induces significantly more bone formation than that induced by the delivery of any single factor or a combination of any two factors.

The scaffolds were delivered to a critical-sized mandibular defect created in 32 Sprague Dawley rats. Epidermal ablation of Dlx3 is linked to IL—associated skin inflammation. In an effort to understand the role of Distal-less 3 Dlx3 in cutaneous biology and pathophysiology, we generated and characterized a mouse model with epidermal ablation of Dlx3.

Thus, Dlx3 ablation in epidermis is linked to altered epidermal differentiation, barrier development, and IL—associated skin inflammation. This model provides a platform that will allow the systematic exploration of the contributions of keratinocytes to cutaneous inflammation.

Suppression of alpha-tocopherol ether- linked acetic acid in VEGF -induced angiogenesis and the possible mechanisms in human umbilical vein endothelial cells. Discontinuation of anti- VEGF cancer therapy promotes metastasis through a liver revascularization mechanism. The impact of discontinuation of anti- VEGF cancer therapy in promoting cancer metastasis is unknown. Here we show discontinuation of anti- VEGF treatment creates a time-window of profound structural changes of liver sinusoidal vasculatures, exhibiting hyper-permeability and enlarged open-pore sizes of the fenestrated endothelium and loss of VE-cadherin.

The drug cessation caused highly leaky hepatic vasculatures permit tumour cell intravasation and extravasation. Discontinuation of an anti- VEGF antibody-based drug and sunitinib markedly promotes liver metastasis. Mechanistically, host hepatocyte, but not tumour cell-derived vascular endothelial growth factor VEGF , is responsible for cancer metastasis.

These findings provide mechanistic insights on anti- VEGF cessation-induced metastasis and raise a new challenge for uninterrupted and sustained antiangiogenic therapy for treatment of human cancers. Pro- and antiangiogenic VEGF and its receptor status for the severity of diabetic retinopathy. Purpose Alteration of pro- and antiangiogenic homeostasis of vascular endothelial growth factor VEGF isoforms in patients with hyperglycemia seems crucial but substantially unexplored at least quantitatively for diabetic retinopathy DR.

Methods The study included participants diabetic retinopathy: 81, diabetic control: 20, non-diabetic control: 22 from the Regional Institute of Ophthalmology, Kolkata. Vascular endothelial growth factor VEGF plays a critical role during normal embryonic angiogenesis and also in the pathological angiogenesis that occurs in a number of diseases, including cancer. Initial attempts to block VEGF by using a humanized monoclonal antibody are beginning to show promise in human cancer patients, underscoring the importance of optimizing VEGF blockade.

Previous studies have found that one of the most effective ways to block the VEGF -signaling pathway is to prevent VEGF from binding to its normal receptors by administering decoy-soluble receptors. The highest-affinity VEGF blocker described to date is a soluble decoy receptor created by fusing the first three Ig domains of VEGF receptor 1 to an Ig constant region; however, this fusion protein has very poor in vivo pharmacokinetic properties.

By determining the requirements to maintain high affinity while extending in vivo half life, we were able to engineer a very potent high-affinity VEGF blocker that has markedly enhanced pharmacokinetic properties.

This VEGF -Trap effectively suppresses tumor growth and vascularization in vivo, resulting in stunted and almost completely avascular tumors.

Sixty-seven women undergoing IVF-ET at a university tertiary hospital were recruited for a prospective study. In serum, concentrations of all the three growth factors were positively correlated with the rate of good quality embryo, and with one another. Concentrations of all the three growth factors in serum were positively correlated with embryo quality, but only serum concentrations of EG- VEGF were associated with the pregnancy outcome. VEGF -A is increased in exogenous endophthalmitis.

Exogenous endophthalmitis is an ophthalmologic emergency defined by panocular inflammation. Vascular endothelial growth factor A VEGF -A contributes to inflammation by promoting chemotaxis of monocytes and granulocytes and by increasing vascular permeability. The purpose of this article is to determine if VEGF -A is elevated in the vitreous samples obtained from individuals with exogenous endophthalmitis. This observation provides a foundation for future studies of targeted VEGF -A blockade in the management of endophthalmitis.

Published by Elsevier Inc. All rights reserved. Endometrial Ablation. If ablation does not control heavy bleeding, further treatment or surgery may be needed. In most cases, women with heavy bleeding The vascular endothelial growth factor VEGF has a strong influence in tumor-related angiogenesis having association with the grade of angiogenesis and the prognosis of different solid tumors including pancreatic cancer.

The genotype of VEGF gene polymorphisms was determined in both patients and controls using polymerase chain reaction-restriction fragment length polymorphism method. The serum VEGF protein was estimated by standard enzyme- linked immunosorbent assay. Serum VEGF level was found to be significantly high in patients Further, the haplotype C-G-T showed a strong association with the disease, and no specific haplotype was associated with increased serum VEGF level.

Published by Elsevier B. The cell lines were inoculated into the subserosal layer of the stomach and subcutaneous tissue of nude mice.

Newly formed lymphatic vessels were detected not only in the periphery but also in the center of the tumors. The intratumor lymphatic vessels connected with the preexisting lymphatic vessels in the muscularis mucosa. The family of vascular endothelial growth factors VEGFs are known for their regulation of vascularization. In the brain, VEGFs are important regulators of angiogenesis, neuroprotection and neurogenesis. Dysregulation of VEGFs is involved in a large number of neurodegenerative diseases and acute neurological insults, including stroke.

Stroke is the main cause of acquired disabilities, and normally results from an occlusion of a cerebral artery or a hemorrhage, both leading to focal ischemia. Neurons in the ischemic core rapidly undergo necrosis. Cells in the penumbra are exposed to ischemia, but may be rescued if adequate perfusion is restored in time.

However, contradictory to what one might expect, endogenously upregulated levels of VEGF as well as the administration of exogenous VEGF is detrimental in acute stroke. This is probably due to VEGF -mediated blood—brain-barrier breakdown and vascular leakage, leading to edema and increased intracranial pressure as well as neuroinflammation.

The present review discusses the multifaceted action of VEGFs in stroke prevention and therapy. Mmigration of undifferentiated neural progenitors is critical for the development and repair of the nervous system. However, the mechanisms and factors that regulate migration are not well understood.

Here, we show that vascular endothelial growth factor VEGF -A, a major angiogenic factor, guides the directed migration of neural progenitors that do not display antigenic markers for neuron- or glia-restricted precursor cells. These results demonstrate that VEGF is an attractive guidance cue for the migration of undifferentiated neural progenitors and offer a mechanistic link between neurogenesis and angiogenesis in the nervous system.

Thromboxane synthase expression and correlation with VEGF and angiogenesis in non-small cell lung cancer. Thromboxane synthase TXS metabolizes prostaglandin H2 into thromboxanes, which are biologically active on cancer cells. TXS over-expression has been reported in a range of cancers, and associated with angiogenesis and poor outcome.

Expression was correlated with a range of clinical parameters, including overall survival. Stable TXS clones were generated and the effect of overexpression on tumor growth and angiogenesis markers was examined in-vitro and in-vivo xenograft mouse model. Molecular characterization of EG- VEGF -mediated angiogenesis: differential effects on microvascular and macrovascular endothelial cells. Endocrine gland derived vascular endothelial growth factor EG- VEGF also called prokineticin PK1 , has been identified and linked to several biological processes including angiogenesis.

Here we characterized its angiogenic effect using different experimental procedures.

Kari byon naked

Kari byon naked

Kari byon naked